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Chemical Tobacco Smoke Pollution

These are exposure studies associated with the chemical and all of its children.

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1–50 of 87 results.
  Reference Associated Study Title Author's Summary Study Factors Stressor Receptors Country Medium Exposure Marker Measurements Outcome
1. Robinson O, et al. (2015). INfancia y Medio Ambiente (INMA) Project In summary, the correlation analysis presented here of multiple environmental exposures among pregnant women provide a first picture of the structure of the exposome during the crucial in utero period. 2,2',3,4,4',5',6-heptabromodiphenyl ether | 2,2',3',4,4',5-hexachlorobiphenyl | 2,2',4,4',5-brominated diphenyl ether | 2,2',4,4'-tetrabromodiphenyl ether | 2,4,5,2',4',5'-hexachlorobiphenyl | Antimony | Arsenic | Benzene | beta-hexachlorocyclohexane | bisphenol A | Cadmium | Cesium | Chloroform | Cobalt | Copper | Cotinine | decabromobiphenyl ether | Dichlorodiphenyl Dichloroethylene | hexabrominated diphenyl ether 153 | hexabromodiphenyl ether 154 | Hexachlorobenzene | Lead | Mercury | Molybdenum | mono(2-ethyl-5-hydroxyhexyl) phthalate | mono(2-ethyl-5-oxohexyl)phthalate | mono-(2-ethylhexyl)phthalate | mono-benzyl phthalate | monobutyl phthalate | monoethyl phthalate | mono-isobutyl phthalate | Nickel | Nitric Oxide | Nitrogen Dioxide | Nitrogen Oxides | Particulate Matter | PCB 180 | pentabrominated diphenyl ether 100 | pentabromodiphenyl ether | perfluorohexanesulfonic acid | perfluorononanoic acid | perfluorooctane sulfonic acid | perfluorooctanoic acid | Pesticides | Phthalic Acids | Selenium | Thallium | Tobacco Smoke Pollution | Trihalomethanes | Zinc Pregnant females Spain air | blood, cord | colostrum | serum | urine | water 2,2',3,4,4',5',6-heptabromodiphenyl ether | 2,2',3',4,4',5-hexachlorobiphenyl | 2,2',4,4',5-brominated diphenyl ether | 2,2',4,4'-tetrabromodiphenyl ether | 2,4,5,2',4',5'-hexachlorobiphenyl | Antimony | Arsenic | Benzene | beta-hexachlorocyclohexane | bisphenol A | Cadmium | Cesium | Chloroform | Cobalt | Copper | Cotinine | decabromobiphenyl ether | Dichlorodiphenyl Dichloroethylene | hexabrominated diphenyl ether 153 | hexabromodiphenyl ether 154 | Hexachlorobenzene | Lead | Mercury | Molybdenum | mono(2-ethyl-5-hydroxyhexyl) phthalate | mono(2-ethyl-5-oxohexyl)phthalate | mono-(2-ethylhexyl)phthalate | mono-benzyl phthalate | monobutyl phthalate | monoethyl phthalate | mono-isobutyl phthalate | Nickel | Nitric Oxide | Nitrogen Dioxide | Nitrogen Oxides | Particulate Matter | PCB 180 | pentabrominated diphenyl ether 100 | pentabromodiphenyl ether | perfluorohexanesulfonic acid | perfluorononanoic acid | perfluorooctane sulfonic acid | perfluorooctanoic acid | Pesticides | Phthalic Acids | Selenium | Thallium | Tobacco Smoke Pollution | Trihalomethanes | Zinc Details
2. Pedersen M, et al. (2012). Dietary exposure to acrylamide was associated with reduced birth weight and head circumference. Consumption of specific foods during pregnancy was associated with higher acrylamide exposure in utero diet Acrylamide | Tobacco Smoke Pollution Fetuses | Pregnant females Denmark|Greece|
Norway|Spain|
United Kingdom
blood, cord Acrylamide | Ethylene Oxide | glycidamide Details Birth Weight | Fetal Growth Retardation | Prenatal Exposure Delayed Effects | head development
3. Perera F, et al. (2008). The findings indicate that neurobehavioral development in Tongliang children benefited by elimination of polycyclic aromatic hydrocarbons (PAH) exposure from the coal-burning plant, consistent with the significant reduction in PAH-DNA adducts in cord blood of children in the 2005 cohort. Air Pollutants | Lead | Mercury | Polycyclic Aromatic Hydrocarbons | Tobacco Smoke Pollution Infants or newborns China blood, cord Lead | Mercury | polycyclic aromatic hydrocarbons-DNA adduct | Tobacco Smoke Pollution Details Prenatal Exposure Delayed Effects | cognition | motor activity
4. Olsson D, et al. (2012). The aim of this study was to assess the association between exposure to ozone and duration of gestational and preterm delivery as well as the association with vehicle exhaust, using nitrogen dioxide as indicator, during different windows of gestation in the Greater Stockholm area. Air Pollutants | Nitrogen Dioxide | Ozone | Tobacco Smoke Pollution Pregnant females Sweden air Nitrogen Dioxide | Ozone Details Premature Birth
5. Wang Z, et al. (2005). In the present study, small expression alerations in several genes, caused by short-term occupational exposure to metal particulates, could be detected in whole-blood total RNA by paired t-tests. tobacco Air Pollutants, Occupational | Tobacco Smoke Pollution Workers United States Particulate Matter Details gene expression
6. Bloom MS, et al. (2014). These results suggest no increased risk for spontaneous pregnancy loss in association with low to moderate level drinking water inorganic arsenic exposure. tobacco Arsenic | Tobacco Smoke Pollution Controls for disease:Abortion, Spontaneous | Subjects with disease:Abortion, Spontaneous | Pregnant females Romania water Arsenic Details Abortion, Spontaneous
7. Alavanja MC, et al. (2004). Agricultural Health Study (AHS) We also found that four heavily used agricultural pesticides, including two herbicides (i.e., metolachlor and pendimethalin) and two insecticides (i.e., chlorpyrifos and diazinon), were associated with a significant excess lung cancer risk in the Agricultural Health Study cohort. Asbestos | Carbofuran | Chlorpyrifos | Diazinon | Dicamba | Dieldrin | Lead | metolachlor | pendimethalin | Tobacco Smoke Pollution Study subjects | Workers United States Details Agricultural Workers' Diseases | Lung Neoplasms
8. Inamura K, et al. (2014). In the present study, lung adenocarcinomas, for which asbestos-exposure and smoking-exposure data could be obtained, were examined for loss of heterozygosity and the p53 mutation. tobacco Asbestos | Tobacco Smoke Pollution Subjects with disease:Adenocarcinoma of lung Japan Details DNA metabolic process
9. Fano V, et al. (2004). Results from this case-control study indicate that the increased risk for lung cancer observed in an Italian area can partially be explained by tobacco smoke and occupational exposures. tobacco Asbestos | Tobacco Smoke Pollution Controls for disease:Lung Neoplasms | Subjects with disease:Lung Neoplasms | Workers Italy Details Lung Neoplasms
10. Reid A, et al. (2005). In this cohort of former workers and residents of Wittenoom, asbestosis is not a mandatory precursor for asbestos related lung cancer. tobacco Asbestos, Crocidolite | Tobacco Smoke Pollution Study subjects | Workers Australia Details Lung Neoplasms
11. Ferrari M, et al. (2015). The short-term use of the specific brand of nicotine-free e-cigarette assessed in this study had no immediate adverse effects on non-smokers and only small effects on forced expiratory volume in 1 s and forced expiratory flow 25% in smokers. tobacco Benzoic Acid | ethyl carbonate | Nicotine | Propylene Glycol | quinoline | Tobacco Smoke Pollution Study subjects Italy Details respiratory gaseous exchange
12. Do MT, et al. (2005). National Enhanced Cancer Surveillance System (NECSS) In the present study we found no evidence of an association between exposure to chlorination disinfection by-products in drinking water and the risk of pancreatic cancer. tobacco bromodichloromethane | Chloroform | Tobacco Smoke Pollution | Trihalomethanes Controls for disease:Pancreatic Neoplasms | Subjects with disease:Pancreatic Neoplasms Canada water bromodichloromethane | Chloroform | Trihalomethanes Details Pancreatic Neoplasms
13. Scinicariello F, et al. (2015). National Health and Nutrition Examination Survey (NHANES) This is the first study reporting an association of blood cadmium levels with depressive symptoms using a nationally representative sample. tobacco Cadmium | Lead | Tobacco Smoke Pollution Study subjects United States blood Cadmium | Lead Details Depressive Disorder
14. Yang H, et al. (2015). Male Reproductive Health in Chongqing College students (MARHCS) The primary objectives of the MARHCS study are to investigate the associations of male reproductive health in young adults with sociopsychobehavioral factors, as well as changes of environmental exposure due to the relocation from rural campus (in University Town) to metro-campus (in central downtown). diet | tobacco Caffeine | Tobacco Smoke Pollution Study subjects China semen Details spermatogenesis | sperm motility
15. Silverman DT, et al. (2012). Diesel Exhaust in Miners Study (DEMS) Our results provide further evidence supporting a causal effect of diesel exhaust exposure on lung cancer mortality in humans. disease | tobacco Carbon | Tobacco Smoke Pollution | Vehicle Emissions Workers United States Carbon Details Death | Lung Neoplasms
16. Yeatts KB, et al. (2012). This study provides new information regarding potential health risks from pollutants commonly found in indoor environments in the United Arab Emirates and other countries. Carbon Monoxide | Formaldehyde | Hydrogen Sulfide | Nitrogen Dioxide | Particulate Matter | Sulfur Dioxide | Tobacco Smoke Pollution Study subjects United Arab Emirates air Carbon Monoxide | Formaldehyde | Hydrogen Sulfide | Nitrogen Dioxide | Particulate Matter | Sulfur Dioxide | Tobacco Smoke Pollution Details Asthma | Respiratory Sounds
17. Joubert BR, et al. (2012). Norwegian Mother and Child Cohort Study (MoBa) We identified a set of genes with methylation changes present at birth in children whose mothers smoked during pregnancy; our findings implicate epigenetic mechanisms in the pathogenesis of the adverse health outcomes associated with this important in utero exposure. tobacco Cotinine | Tobacco Smoke Pollution Infants or newborns | Pregnant females Norway blood, cord | plasma AHRR | CNTNAP2 | Cotinine | CYP1A1 | EXT1 | GFI1 | HLA-DPB2 | MYO1G | RUNX1 | TTC7B Details Prenatal Exposure Delayed Effects | DNA methylation on cytosine within a CG sequence | regulation of DNA methylation
18. Lai PS, et al. (2016). Shanghai Textile Worker Study In this workplace-based cohort of occupational organic dust and smoking exposure, we find that occupational endotoxin exposure is associated with persistent increases in lung density, lung mass, and airway thickening even after exposure cessation. Endotoxins | Tobacco Smoke Pollution Workers China Details Lung Diseases | lung growth
19. Antoniewicz L, et al. (2016). In healthy volunteers, ten puffs of e-cigarette vapor inhalation caused an increase in endothelial progenitor cells. Ethanol | Glycerol | Nicotine | Propylene Glycol | Tobacco Smoke Pollution Study subjects Sweden breath | plasma Cotinine | Nitric Oxide Details stem cell population maintenance
20. Shirinde J, et al. (2014). Children living in one of the air pollution priority areas of South Africa have an increased risk of wheezing due to exposure to both indoor and outdoor air pollution sources. Fuel Oils | Kerosene | Tobacco Smoke Pollution | Vehicle Emissions Children South Africa Details Respiratory Sounds
21. Vuong AM, et al. (2016). Health Outcomes and Measures of the Environment (HOME) Maternal serum concentrations of polybrominated diphenyl ethers during early pregnancy may alter measures of child anthropometry. Halogenated Diphenyl Ethers | hexabrominated diphenyl ether 153 | pentabrominated diphenyl ether 100 | Tobacco Smoke Pollution Infants or newborns | Pregnant females United States serum Halogenated Diphenyl Ethers Details developmental growth | lipid biosynthetic process
22. Mervish NA, et al. (2016). Breast Cancer and the Environment Research Project (BCERP) | National Health and Nutrition Examination Survey (NHANES) We examined whether exposure to sodium/iodide symporter inhibitors measured at one time point were associated with height, waist circumference, and body mass index trajectories during childhood in an established cohort group of young girls with adequate iodine intake. Iodides | Nitrates | perchlorate | thiocyanate | Tobacco Smoke Pollution Children United States urine Iodides | Nitrates | perchlorate | thiocyanate Details developmental growth
23. Braun JM, et al. (2008). National Health and Nutrition Examination Survey (NHANES) These results suggest that prenatal tobacco exposure and environmental lead exposure contribute substantially to conduct disorder in U.S. children. Lead | Tobacco Smoke Pollution Children United States blood | serum Cotinine | Lead Details Conduct Disorder
24. Sexton K, et al. (2012). School Health Initiative Environment, Learning, and Disease (SHIELD) Biologic markers confirm that children from a low-income, ethnically diverse neighborhood experienced concurrent exposure to a variety of hazardous environmental chemicals during their everyday activities. socioeconomic status Metals, Heavy | Organothiophosphorus Compounds | Pesticides | Phthalic Acids | Polychlorinated Biphenyls | Tobacco Smoke Pollution | Volatile Organic Compounds Children United States blood | urine 2,2',3',4,4',5-hexachlorobiphenyl | 2,2',3,5,5',6-hexachlorobiphenyl | 2,3,3',4,4',5-hexachlorobiphenyl | 2,3',4,4',5-pentachlorobiphenyl | 2,4,4',5-tetrachlorobiphenyl | 2,4,5,2',4',5'-hexachlorobiphenyl | 2-xylene | beta-hexachlorocyclohexane | Cotinine | DDT | Dichlorodiphenyl Dichloroethylene | dimethyl phosphate | Lead | mono(2-ethyl-5-hydroxyhexyl) phthalate | mono(2-ethyl-5-oxohexyl)phthalate | mono-(2-ethylhexyl)phthalate | mono-benzyl phthalate | nonachlor | PCB 180 | Tetrachloroethylene | Toluene Details
25. Flouris AD, et al. (2013). In this study, we present the first comprehensive and standardized assessment regarding the impact of short term active and passive e-cigarette smoking on cotinine concentration and lung function compared to active and passive tobacco cigarette smoking. tobacco Nicotine | Tobacco Smoke Pollution Study subjects Greece serum Cotinine Details respiratory gaseous exchange
26. Vinnikov D, et al. (2010). In summary, we found levels of exposure to secondhand smoke both in socializing venues and at home to be high in participants in this study conducted in Kyrgyzstan. tobacco Nicotine | Tobacco Smoke Pollution Workers Kyrgyzstan air | hair Nicotine Details Respiration Disorders
27. Peters KO, et al. (2017). Center for Childhood Asthma in the Urban Environment (CCAUE) Time spent in non-smoking homes was associated with significantly decreased urine 1-hydroxypyrene-glucuronide (1-OHPG), and secondhand smoke exposures increased these levels; time spent outdoors was associated with increased urinary 1-OHPG concentrations in boys only; our results suggest that secondhand smoke and ambient outdoor air pollution contribute to internal dose of polycyclic aromatic hydrocarbons in inner city children. sex | tobacco Particulate Matter | Polycyclic Aromatic Hydrocarbons | Tobacco Smoke Pollution Children United States air, indoor | urine 1-hydroxypyrene-glucuronide | Nicotine | Particulate Matter Details
28. Baccarelli AA, et al. (2016). In this nationwide analysis of ~28 million individuals aged 55+ across 3,034 counties, higher levels of PM2.5 air pollution were associated with lower population-based probabilities of exceptional aging, even after adjusting for smoking, obesity, demographic and socioeconomic variables, total and age-specific migration rates, and differences across the nine census divisions of the U.S. body mass index | socioeconomic status | tobacco Particulate Matter | Tobacco Smoke Pollution Study subjects United States Particulate Matter Details Death
29. Migliaccio CT, et al. (2009). These results demonstrate that levoglucosan in the lungs is detectable in the urine of both mice and humans and that it is a good candidate as a biomarker of exposure to biomass smoke. Particulate Matter | Tobacco Smoke Pollution Children United States air | urine 1,6-anhydro-beta-glucopyranose | Cotinine | Particulate Matter Details
30. Mucha AP, et al. (2006). Environmental Pollutants and Health Status of Children We measured urinary 1-hydroxypyrene (1-OHP) in 48 children 3 years of age in Mariupol, Ukraine, who lived near a steel mill and coking facility and compared these with 1-OHP concentrations measured in 42 children of the same age living in the capital city of Kiev, Ukraine. Polycyclic Aromatic Hydrocarbons | Tobacco Smoke Pollution Children Ukraine air | urine 1-hydroxypyrene | Benzo(a)pyrene | pyrene Details
31. Polanska K, et al. (2014). Polish Mother and Child Cohort Study This study provides evidence that combined exposure of pregnant women to common environmental pollutants such as polycyclic aromatic hydrocarbons and environmental tobacco smoke might adversely affect fetal development. Polycyclic Aromatic Hydrocarbons | Tobacco Smoke Pollution Infants or newborns | Pregnant females Poland urine 1-hydroxyphenanthrene | 1-hydroxypyrene | 4-hydroxyphenanthrene | 9-phenanthrol | Phenanthrenes | Pyrenes Details developmental growth
32. White AJ, et al. (2016). Long Island Breast Cancer Study Project (LIBCSP) This study considers the impact on breast cancer incidence from multiple polycyclic aromatic hydrocarbons exposure sources in a single model, which better reflects exposure to these complex mixtures; groups of sources, particularly indoor sources, were associated with an increase in breast cancer incidence. tobacco Polycyclic Aromatic Hydrocarbons | Tobacco Smoke Pollution | Vehicle Emissions Subjects with disease:Breast Neoplasms United States Details Breast Neoplasms
33. Perera FP, et al. (2007). These results suggest that exposure to elevated levels of polycyclic aromatic hydrocarbons (PAHs), indicated by PAH-DNA adducts in cord blood, may have contributed to a modest reduction in cognitive development among children prenatally exposed to the World Trade Center event. tobacco polycyclic aromatic hydrocarbons-DNA adduct | Tobacco Smoke Pollution Children United States Details cognition
34. Richter PA, et al. (2013). National Health and Nutrition Examination Survey (NHANES) Youths with secondhand smoke exposure had blood lead levels suggestive of the potential for adverse cognitive outcomes. Tobacco Smoke Pollution Children | Study subjects United States blood Lead Details
35. Baheiraei A, et al. (2014). Maternal environmental tobacco smoking exposure affects milk lipids which are essential for infant growth. Tobacco Smoke Pollution Mothers Iran, Islamic Republic of milk, human Cotinine Details lipid metabolic process
36. Breton CV, et al. (2014). Childhood Asthma Management Program (CAMP) Our data suggest that prenatal exposure to tobacco smoke is associated with reproducible epigenetic changes that persist well into childhood; however, the biological significance of these altered loci remains unknown. tobacco Tobacco Smoke Pollution Subjects with disease:Asthma | Children United States blood cell C11ORF52 | FRMD4A Details DNA methylation on cytosine within a CG sequence
37. Li YF, et al. (2014). Interactions between single-nucleotide polymorphisms in the intercellular adhesion molecule 1 (ICAM1) gene and environmental tobacco smoke are likely to be involved in the development of asthma in childhood. genetics | tobacco Tobacco Smoke Pollution Controls for disease:Asthma | Subjects with disease:Asthma | Children | Subjects with gene influence:ICAM1 Taiwan, Province of China Details Asthma
38. Eze IC, et al. (2014). Swiss study on Air Pollution and Lung Diseases In Adults (SAPALDIA) We found a positive association between environmental tobacco smoke exposure and diabetes mellitus in never smokers, which was significantly stronger in older subjects and in subjects with chronic obstructive pulmonary disease. age | disease Tobacco Smoke Pollution Study subjects Sweden Details Diabetes Mellitus
39. Lee KW, et al. (2015). Saguenay Youth Study (SYS) The results of the present study suggest that prenatal exposure to maternal cigarette smoking may interact with OPRM1 to enhance dietary intake of fat, and that the mechanisms of this gene-environment interaction might involve exposure-associated decreases of gene-body DNA methylation in OPRM1. Tobacco Smoke Pollution Children | Subjects with gene influence:OPRM1 Canada Details Obesity | DNA methylation | feeding behavior
40. Gustavsen MW, et al. (2014). In this Norwegian MS case-control study of environmental exposures, we replicate that infectious mononucleosis and smoking are associated with increased MS risk. tobacco Tobacco Smoke Pollution Controls for disease:Multiple Sclerosis | Subjects with disease:Multiple Sclerosis Norway Details Multiple Sclerosis
41. Hedström AK, et al. (2014). Epidemiological Investigation of Multiple Sclerosis (EIMS) | Kaiser Permanente Medical Care Plan (KPNC) The risk of developing multiple sclerosis associated with different HLA genotypes may be influenced by exposure to passive smoking. genetics | tobacco Tobacco Smoke Pollution Subjects with gene influence:HLA-A | Subjects with gene influence:HLA-DRB1 | Controls for disease:Multiple Sclerosis | Subjects with disease:Multiple Sclerosis Sweden Details Multiple Sclerosis
42. Kamer B, et al. (2014). The results demonstrate that 17% of all the examined children were those exposed to tobacco smoke effects already in their fetal life; moreover, it was observed that the young age and lower education level of their parents, together with poor housing conditions, may suggest a predisposing character. socioeconomic status | tobacco Tobacco Smoke Pollution Children Poland urine Cotinine Details
43. Eaton JE, et al. (2015). PSC Resource of Genetic Risk, Environment and Synergy Studies (PROGRESS) We performed a large, prospective multicenter study with two key aims: (i) identify novel exposures associated with primary sclerosing cholangitis (PSC) among those with and without inflammatory bowel disease (IBD); (ii) validate or refute environmental exposures that could potentially be associated with PSC (regardless of IBD status) that have been described in previous studies. diet | disease | tobacco Tobacco Smoke Pollution Subjects with disease:Cholangitis, Sclerosing | Subjects with disease:Inflammatory Bowel Diseases Canada|United States Details Cholangitis, Sclerosing | Inflammatory Bowel Diseases
44. Zhang Y, et al. (2016). ESTHER Study Our study strongly supports the potential utility of DNA methylation markers as indicators for both current and lifetime smoking exposure and for predicting mortality outcomes, in particular for cardiovascular mortality. Tobacco Smoke Pollution Study subjects Germany Details Cardiovascular Diseases | Death | Neoplasms | DNA methylation
45. Rosen L, et al. (2015). In a pilot study involving 27 smoking families (two parents, one child), a Sidepak was used to continuously monitor home particulate matter (PM2.5) during an 8-hour period, and provided real-time feedback; improvement in tobacco smoke measurement in the home is needed to encourage and help maintain smoke-free homes and protect vulnerable children. tobacco Tobacco Smoke Pollution Study subjects Israel air Particulate Matter Details
46. Tran TT, et al. (2015). Thai Cohort Study (TCS) A high proportion of cohort members, especially females, were passive smokers at home and at public transport stations; males were more exposed at workplace and recreational places; we observed a social gradient with more passive smoking in poorer people; we also observed a dose response relationship linking graded smoke exposures to chronic conditions, such as less well-being and more psychological distress. sex | socioeconomic status | tobacco Tobacco Smoke Pollution Study subjects Thailand Details Chronic Disease
47. Moore BF, et al. (2016). National Health and Nutrition Examination Survey (NHANES) Interaction results suggest that the prevalence of obesity among children with both high exposure to secondhand smoke and low levels of certain nutrients (dietary fiber, eicosapentaenoic acid, or docosahexaenoic acid) is greater than would be expected due to the effects of the individual exposures alone. age | diet | tobacco Tobacco Smoke Pollution Children United States serum | urine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butan-1-ol | Cotinine Details Pediatric Obesity
48. Rafiq R, et al. (2016). Our findings indicate increased risk of esophageal squamous cell carcinoma due to secondhand smoking exposure in dose-dependent manner. socioeconomic status | tobacco Tobacco Smoke Pollution Controls for disease:Esophageal Squamous Cell Carcinoma | Subjects with disease:Esophageal Squamous Cell Carcinoma India Details Esophageal Squamous Cell Carcinoma
49. Zhu X, et al. (2016). Wuhan-Zhuhai (WHZH) cohort We identified DNA methylation markers associated with smoking in a Chinese population, including some markers that were also correlated with gene expression. tobacco Tobacco Smoke Pollution Study subjects China urine 1-hydroxyphenanthrene | 1-hydroxypyrene | 1-naphthol | 2-hydroxyfluorene | 2-naphthol | 4-hydroxyphenanthrene | 9-phenanthrol | fluoren-9-ol | Polycyclic Aromatic Hydrocarbons Details DNA methylation on cytosine within a CG sequence
50. Ambatipudi S, et al. (2016). European Prospective Investigation into Cancer and Nutrition (EPIC) Study Our study has comprehensively cataloged the smoking-associated DNA methylation alterations and showed that these alterations are reversible after smoking cessation. tobacco Tobacco Smoke Pollution Study subjects Denmark|France|
Germany|Greece|
Italy|Netherlands|
Norway|Spain|
Sweden|United Kingdom
Details regulation of DNA methylation
1–50 of 87 results.