These are exposure studies associated with the chemical and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Wood SL, et al. (2007).||There appears to be no relationship between serum concentrations of Dichlorodiphenyl Dichloroethylene, daidzein, and genistein and spontaneous preterm labor in our population.||daidzein | Dichlorodiphenyl Dichloroethylene | Genistein||Pregnant females | Controls for disease:Premature Birth | Subjects with disease:Premature Birth||Canada||serum||daidzein | Dichlorodiphenyl Dichloroethylene | Genistein||Details|
|2.||Teitelbaum SL, et al. (2008).||We evaluated intraindividual temporal variability in 19 urinary biomarkers of endocrine disrupters (eight phthalate metabolites from six phthalate diesters, six phytoestrogens, and five phenols) among New York City minority children.||Endocrine Disruptors||Children||United States||urine||2,3-
|3.||Harlid S, et al. (2016).||Infant Feeding and Early Development (IFED) Study||Girls fed soy formula have altered DNA methylation in vaginal cell DNA which may be associated with decreased expression of an estrogen-responsive gene.||Genistein||Infants or newborns||cell, epithelial||BTBD11 | MAS1L | PRR5L||Details||DNA methylation|
|4.||Wolff MS, et al. (2015).||Breast Cancer and the Environment Research Project (BCERP)||We estimated relative risk of age at puberty associated with phenol exposures among girls when they were 6-8 years old and followed annually for 7 years; girls experienced breast development 5-6 months later (for enterolactone and benzophenone-3) or 4-9 months sooner (for triclosan and 2,5-dichlorophenol).||sex||Phenols||Children||United States||urine||2,3-