These are exposure studies associated with the disease and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Xue X, et al. (2013).||The hOGG1 Ser326Cys polymorphism might be associated with the risk of lung adenocarcinoma in Chinese non-smoking females. Furthermore, there is a significant gene-environment association between cooking oil fumes and hOGG1 326 Cys/Cys genotype in lung adenocarcinoma among female non-smokers.||genetics||Air Pollutants||Controls for disease:Adenocarcinoma of lung | Subjects with disease:Adenocarcinoma of lung | Subjects with gene influence:OGG1||China||Details||Adenocarcinoma of lung|
|2.||Inamura K, et al. (2014).||In the present study, lung adenocarcinomas, for which asbestos-exposure and smoking-exposure data could be obtained, were examined for loss of heterozygosity and the p53 mutation.||tobacco||Asbestos | Tobacco Smoke Pollution||Subjects with disease:Adenocarcinoma of lung||Japan||Details||DNA metabolic process|
|3.||Tual S, et al. (2016).||Agricultural Health Study (AHS)||This study provides preliminary evidence of an increased risk of lung adenocarcinoma among daily drivers of diesel tractors and suggests that exposure to endotoxins may modify the impact of diesel exposure on lung cancer risk.||Vehicle Emissions||Study subjects | Workers||United States||Details||Adenocarcinoma of lung|