These are exposure studies associated with the disease and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Cave M, et al. (2010).||National Health and Nutrition Examination Survey (NHANES)||In the present study, we tested the hypothesis that environmental pollutants are dose-dependently associated with increased risk for alanine aminotransferase elevation and suspected nonalcoholic fatty liver disease in the NHANES adult population.||2,2',3,3',4,4',5-
||Study subjects||United States||blood | urine||2,2',3,3',4,4',5-
|2.||Frediani JK, et al. (2018).||National Health and Nutrition Examination Survey (NHANES)||Our findings demonstrate a positive association between urinary arsenic exposure and risk of nonalcoholic fatty liver disease among U.S. adolescents and adults that is highest among Mexican Americans and among those obese, regardless of race/ethnicity.||disease | race||Arsenic||Study subjects||United States||urine||Arsenic||Details||Non-alcoholic Fatty Liver Disease | transaminase activity|
|3.||Hyder O, et al. (2013).||National Health and Nutrition Examination Survey (NHANES)||Environmental cadmium exposure was associated with hepatic necroinflammation, non-alcoholic fatty liver disease, and non-alcoholic steatohepatitis in men, and hepatic necroinflammation in women.||age | race | sex||Cadmium||Study subjects||United States||urine||Cadmium||Details||Death | Liver Diseases | Non-alcoholic Fatty Liver Disease | inflammatory response | necrotic cell death|