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Disease Inflammation

These are exposure studies associated with the disease and all of its children.

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1–11 of 11 results.
  Reference Associated Study Title Author's Summary Study Factors Stressor Receptors Country Medium Exposure Marker Measurements Outcome
1. Jiménez-Garza O, et al. (2017). Our results demonstrate alterations in the inflammation pathway at the epigenetic level associated with exposure to benzene. Acetone | Benzene | ethylbenzene | methyl acetate | methylethyl ketone | n-hexane | Toluene | Xylenes Workers Mexico air Acetone | Benzene | ethylbenzene | methyl acetate | methylethyl ketone | n-hexane | Toluene | Xylenes Details Inflammation | inflammatory response | methylation
2. van den Hooven EH, et al. (2012). Generation R Study Our results suggest that exposure to air pollution during pregnancy may lead to maternal and fetal inflammatory responses. Nitrogen Dioxide | Particulate Matter Infants or newborns | Pregnant females Netherlands air Nitrogen Dioxide | Particulate Matter Details Inflammation | regulation of gene expression
3. Szyszkowicz M, et al. (2010). Increase in concentration of ambient ground-level ozone was associated with increased hazard of emergency department visit for cellulites in distinct age groups and in characteristic environmental conditions enhancing ozone potency. Ozone Canada air Ozone Details Cellulitis
4. Hoffmann B, et al. (2009). German Heinz Nixdorf Recall Study Our study indicates that long-term residential exposure to high levels of Particulate Matter (2.5) is associated with systemic inflammatory markers in men. sex Ozone | Particulate Matter Controls for disease:Coronary Disease | Subjects with disease:Coronary Disease | Controls for disease:Diabetes Mellitus | Subjects with disease:Diabetes Mellitus | Study subjects Germany air CRP | Ozone | Particulate Matter Details Atherosclerosis | Inflammation
5. Barath S, et al. (2013). Exposure to diesel exhaust, but not ozone, increases the concentration of fraction of exhaled nitric oxide (FENO) FENO10 and FENO50 in healthy subjects, suggesting an inflammatory response mainly located in the central airways. Ozone | Vehicle Emissions Study subjects Sweden Details Inflammation
6. Siponen T, et al. (2015). Results suggest that particulate matter (2.5) from several sources, such as biomass combustion and traffic, are promoters of systemic inflammation, a risk factor for cardiovascular diseases. Particulate Matter Subjects with disease:Myocardial Ischemia Finland air Particulate Matter Details Cardiovascular Diseases | Inflammation | inflammatory response
7. Zhao J, et al. (2015). The results indicated that traffic exposure to high levels of Particulate Matter (2.5) was associated with systemic inflammatory response and respiratory injury. Particulate Matter Workers China air | serum CRP | Particulate Matter Details Inflammation | PULMONARY FUNCTION | gene expression | inflammatory response
8. Jacobs L, et al. (2010). Diabetic patients showed evidence of proinflammatory response to both recent and chronic exposure to Particulate Matter air pollution. Particulate Matter Subjects with disease:Diabetes Mellitus Belgium air | sputum Carbon | Particulate Matter Details Inflammation | platelet activation
9. Bernatsky S, et al. (2011). We provide novel data that suggest that short-term variations in air pollution may influence disease activity in established autoimmune rheumatic disease in humans. Particulate Matter Subjects with disease:Lupus Erythematosus, Systemic Canada air Particulate Matter Details Autoimmune Diseases | Inflammation | Lupus Erythematosus, Systemic
10. Chen JC, et al. (2008). National Health and Nutrition Examination Survey (NHANES) Our study revealed a positive association between long-term particulate matter exposure and hematological markers of inflammation and supported the hypothesized metabolic syndrome-dependent susceptibility. disease Particulate Matter Controls for disease:Metabolic Syndrome | Subjects with disease:Metabolic Syndrome | Study subjects United States Particulate Matter Details Inflammation | inflammatory response
11. Fang SC, et al. (2012). Normative Aging Study (NAS) In an elderly male population, estimated black carbon exposures were positively associated with markers of systemic inflammation but only in men with coronary heart disease or diabetes. disease Soot Subjects with disease:Coronary Disease | Subjects with disease:Diabetes Mellitus | Study subjects United States serum CXCL8 | IL1B | IL6 | Soot | TNF | VEGFA Details Inflammation | cytokine production involved in inflammatory response | interleukin-1 beta production | interleukin-6 production | interleukin-8 production | regulation of gene expression | tumor necrosis factor production | vascular endothelial growth factor production
1–11 of 11 results.